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Effect of AS-IV on body weight changes and myocardial injury markers in DOX-treated juvenile mice. (A) Time-course of body weight changes in mice from Day 0 to Day 3. DOX treatment caused significant growth retardation, which was attenuated by AS-IV. (B) Quantitative analysis of <t>serum</t> <t>CK-MB</t> levels in each group. **, P<0.01 vs. control group. AS-IV, astragaloside IV; CK-MB, creatine <t>kinase-muscle/brain;</t> DOX, doxorubicin; PBS, phosphate buffered saline.
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Effect of AS-IV on body weight changes and myocardial injury markers in DOX-treated juvenile mice. (A) Time-course of body weight changes in mice from Day 0 to Day 3. DOX treatment caused significant growth retardation, which was attenuated by AS-IV. (B) Quantitative analysis of <t>serum</t> <t>CK-MB</t> levels in each group. **, P<0.01 vs. control group. AS-IV, astragaloside IV; CK-MB, creatine <t>kinase-muscle/brain;</t> DOX, doxorubicin; PBS, phosphate buffered saline.
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Effect of AS-IV on body weight changes and myocardial injury markers in DOX-treated juvenile mice. (A) Time-course of body weight changes in mice from Day 0 to Day 3. DOX treatment caused significant growth retardation, which was attenuated by AS-IV. (B) Quantitative analysis of <t>serum</t> <t>CK-MB</t> levels in each group. **, P<0.01 vs. control group. AS-IV, astragaloside IV; CK-MB, creatine <t>kinase-muscle/brain;</t> DOX, doxorubicin; PBS, phosphate buffered saline.
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Effect of AS-IV on body weight changes and myocardial injury markers in DOX-treated juvenile mice. (A) Time-course of body weight changes in mice from Day 0 to Day 3. DOX treatment caused significant growth retardation, which was attenuated by AS-IV. (B) Quantitative analysis of <t>serum</t> <t>CK-MB</t> levels in each group. **, P<0.01 vs. control group. AS-IV, astragaloside IV; CK-MB, creatine <t>kinase-muscle/brain;</t> DOX, doxorubicin; PBS, phosphate buffered saline.
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Effect of AS-IV on body weight changes and myocardial injury markers in DOX-treated juvenile mice. (A) Time-course of body weight changes in mice from Day 0 to Day 3. DOX treatment caused significant growth retardation, which was attenuated by AS-IV. (B) Quantitative analysis of serum CK-MB levels in each group. **, P<0.01 vs. control group. AS-IV, astragaloside IV; CK-MB, creatine kinase-muscle/brain; DOX, doxorubicin; PBS, phosphate buffered saline.

Journal: Translational Cancer Research

Article Title: The effect of astragaloside IV on doxorubicin-induced cardiotoxicity in young mice: exploring its cardioprotective effect during childhood tumor chemotherapy

doi: 10.21037/tcr-2026-1-0080

Figure Lengend Snippet: Effect of AS-IV on body weight changes and myocardial injury markers in DOX-treated juvenile mice. (A) Time-course of body weight changes in mice from Day 0 to Day 3. DOX treatment caused significant growth retardation, which was attenuated by AS-IV. (B) Quantitative analysis of serum CK-MB levels in each group. **, P<0.01 vs. control group. AS-IV, astragaloside IV; CK-MB, creatine kinase-muscle/brain; DOX, doxorubicin; PBS, phosphate buffered saline.

Article Snippet: Cell Counting Kit-8 (CCK-8) for cell viability detection, reactive oxygen species (ROS), dichlorodihydrofluorescein diacetate (DCFH-DA) for oxidative stress assessment, and superoxide dismutase (SOD), malondialdehyde (MDA), glutathione (GSH) and creatine kinase-muscle/brain (CK-MB) biochemical detection kits were obtained from Nanjing Jiancheng Bioengineering Institute (Nanjing, China) ( ).

Techniques: Control, Saline